Monday, June 25, 2012

A very interesting new approach to asthma

There's a lot more that's really important, but here's the crux:

Some inhalants patients use to relieve their asthmatic symptoms work by stimulating beta2-adrenergic receptors in the bronchial tubes.[28] Stimulating the receptors dilates the tubes and constricts blood vessels in their lining, which dries mucous secretions. These effects of the inhalants relieve shortness of breath, coughing, and wheezing respiration.[25, p.207]

An alternative to using the inhalers is to increase the number of beta2-adrenergic receptors in the bronchial tubes by taking an effective daily dose of thyroid hormone.[33] T3 is especially useful for this purpose.

In 1991, Egyptian researchers treated 23 asthmatic children with T3.[29] The children weren't hypothyroid. During the 30 days of treatment, they continued to use their usual anti-asthma drugs as needed, but they reduced their doses as low as they could.

The researchers wrote, "They [the children] all reported at the end of the 30 days an obvious subjective improvement of their asthmatic conditions with a decrease in the number of exacerbations. Seven patients stopped their usual anti-asthmatic medicines, being maintained on T3 only and 3 have decreased the amount of bronchodilators needed. A significant improvement of pulmonary function tests was noted in all patients."

According to the researchers, "All patients tolerated well the T3 regimen without any adverse effect." They concluded that T3 induced beneficial effects: T3 "proves to be a useful adjuvant to classic anti-asthma therapy, and may reduce the amount of bronchodilators needed."
Air Hunger to Death: Breathing Problems of Hypothyroid Patients

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